Ik vond nog iets:
http://www.veterinarypracticenews.com/J ... -For-Lyme/In the Lab
Experimentally, adult beagles given Lyme disease did not get sick. They just became Lyme-positive, says Meryl P. Littman, VMD, Dipl. ACVIM, of Penn’s School of Veterinary Medicine
“Less common is Lyme nephropathy, often in Labradors and golden retrievers, which is a serious protein-losing kidney disease that may not respond to antibiotics because it is an immune-mediated disease triggered by exposure to Lyme antigens.
“Signs of other tick-borne diseases can include similar symptoms to Lyme disease. The diagnosis of Lyme is often made too fast, based on a Lyme positive test. The signs may actually be due to a co-infection with one of the many other tick-borne diseases in our area or some other disease process entirely.”
Research shows that dogs testing positive for both Lyme and Anaplasmosis are twice as likely to show clinical symptoms, says Leif Lorentzen, DVM, senior medical affairs manager at Idexx
Lyme in Dogs
Exposure to Lyme disease is common in dogs, but the disease isn’t, Littman says.
http://www.terrierman.com/Lyme-in-Dogs.pdfOf zou hij het interview met het RIVM dat onlangs op Radio 1 was te horen hebben geluisterd?
http://www.nporadio2.nl/nieuws/11610/kr ... e-van-lymeQuote:
"Katten worden doorgaans niet ziek door de bacterie die in teken zit en lyme veroorzaakt bij mensen, en honden bijna nooit.
Heel soms wordt een hond wel eens ziek."
Verder zegt hij:
"Het is soortafhankelijk."
PS.
Wel weer jammer dat ie Borrelia een bacterie noemt, want Borrelia is een spirocheet met zijn eigen stam.
En ik vraag me ook af waar hij dat op baseert, dat katten bv niet ziek kunnen worden, en honden soms.
Dat "Experimentally" waar Meryl P. Littman het over heeft zal wel het vaccin zijn dat is losgelaten op onze huisdieren.
Ook vraag ik me af of er überhaupt goed is gekeken naar neuroborreliose bij honden en katten.
Bij paarden komt in ieder geval wel neuroborreliose voor:
http://vet.sagepub.com/content/48/6/1151.full.pdfAbstract
Lyme neuroborreliosis—characterized as chronic, necrosuppurative to nonsuppurative, perivascular to diffuse meningoradiculoneuritis—was diagnosed in 2 horses with progressive neurologic disease. In 1 horse, Borrelia burgdorferi sensu stricto was identified by polymerase chain reaction amplification of B burgdorferi sensu stricto–specific gene targets (ospA, ospC, flaB, dbpA, arp). Highest spirochetal burdens were in tissues with inflammation, including spinal cord, muscle, and joint capsule. Sequence analysis of ospA, ospC, and flaB revealed 99.9% sequence identity to the respective genes in B burgdorferi strain 297, an isolate from a human case of neuroborreliosis. In both horses, spirochetes were visualized in affected tissues with Steiner silver impregnation and by immunohistochemistry, predominantly within the dense collagenous tissue of the dura mater and leptomeninges.